Vitamin D and Your Teeth: Why the "Sunshine Vitamin" Matters for Gums, Bone and Dental Implant Success

Introduction: The Nutrient Hiding in Plain Sight
Ask most people what keeps teeth healthy and they will say brushing, flossing and avoiding sugar. All true. But there is a factor working underneath the enamel and beneath the gums that rarely makes the list β vitamin D.
Vitamin D is not really a vitamin at all in the strict sense. It behaves like a hormone, produced in the skin under sunlight and switched on by the liver and kidneys before it goes to work in almost every tissue in the body. In the mouth, it governs how much calcium your jaw bone can lay down, how effectively your gums fight bacteria, how quickly a surgical wound closes, and β increasingly the focus of research β whether a dental implant successfully bonds to bone.
At our City of London practice we place implants, treat gum disease and run private blood tests under one roof, so we see both sides of this story: patients whose implants integrate beautifully, and the smaller number whose healing is slower or more complicated than expected. Vitamin D status is one of the modifiable factors that can tip the balance.
This guide is long because the topic deserves it. We cover the biology in plain English, what the UK numbers really mean, the evidence linking vitamin D to gum disease, tooth decay and implant failure, how to test and correct a deficiency, and a practical timeline to follow before implant surgery.
0 in 6
UK adults have low vitamin D across the year
NDNS data; higher in winter
0 Β΅g
Daily supplement advised by the NHS in autumn & winter
400 IU per day
0%+
Typical 10-year implant survival in healthy patients
Systematic reviews
0 nmol/L
Level many surgeons aim for before implant placement
No universal consensus
What Vitamin D Actually Does in the Body
Two forms, one measurement
There are two dietary forms: vitamin D2 (ergocalciferol, from plants and fortified foods) and vitamin D3 (cholecalciferol, made in the skin and found in oily fish, egg yolk and most supplements). D3 is generally more effective at raising and maintaining blood levels.
Whichever form you take in, it travels to the liver and is converted to 25-hydroxyvitamin D β written 25(OH)D. This is the storage form and it is what a blood test measures. From there, the kidneys (and, importantly, many local tissues including bone and immune cells) convert it to the active hormone 1,25-dihydroxyvitamin D, or calcitriol.
How vitamin D reaches your jaw
Sunlight / diet
UVB on skin, oily fish, eggs, supplements
βLiver
Converted to 25(OH)D β the form we measure
βKidney
Activated to 1,25(OH)βD (calcitriol)
βJaw bone & gums
Calcium absorption, bone remodelling, immune defence
A blood test measures 25(OH)D, the storage form β the best indicator of your overall vitamin D status.
The three jobs that matter most for your mouth
- Calcium and phosphate absorption. Without adequate vitamin D the gut absorbs only a fraction of the calcium you eat. Bone β including the alveolar bone that holds your teeth and any implants β cannot mineralise properly without it.
- Bone remodelling. Bone is constantly being broken down by osteoclasts and rebuilt by osteoblasts. Vitamin D helps regulate both, keeping the balance in favour of strong, dense bone.
- Immune modulation. Vitamin D increases the production of antimicrobial peptides such as cathelicidin and defensins in saliva and gum tissue, and dampens excessive inflammatory responses. Both effects are directly relevant to gum disease and to healing around implants.
Bone density
Drives calcium and phosphate absorption so the jaw can mineralise around an implant.
Immune defence
Boosts antimicrobial peptides in saliva and gum tissue; helps control inflammation.
Healing
Supports osteoblast activity and soft-tissue repair after extractions and surgery.
Long-term stability
Linked with lower peri-implantitis risk and healthier gums around natural teeth.
How Common Is Deficiency in the UK?
Very. The UK sits at a latitude where, between roughly October and March, the sun is too low for skin to make meaningful vitamin D regardless of how long you spend outside. Add office-based working patterns, sunscreen use, darker skin tones (which need longer exposure), covering clothing and a diet low in oily fish, and the picture is predictable.
According to the National Diet and Nutrition Survey, around one in six UK adults has a 25(OH)D level below 25 nmol/L across the year, the threshold associated with risk of bone softening. In the winter months that rises sharply, with some studies putting it closer to one in three. Levels in the "insufficient" 25β50 nmol/L band are more common still.
This is why, since 2016, Public Health England (now the Office for Health Improvement and Disparities) has advised every adult and child over one to consider a daily 10 Β΅g (400 IU) supplement during autumn and winter, and year-round for people who are rarely outdoors, cover their skin or have darker skin.
For the City of London specifically β a population that commutes underground, works indoors and often exercises in gyms rather than parks β our clinical experience is that low vitamin D is the rule rather than the exception among patients who have never supplemented.
Who is at particular risk?
- People with darker skin (Black, South Asian, Middle Eastern heritage)
- Office and shift workers with little daytime outdoor exposure
- Anyone who covers most of their skin outdoors
- Older adults (skin becomes less efficient at producing vitamin D)
- People who are overweight (vitamin D is stored in fat and less available)
- Those with malabsorption conditions (coeliac disease, Crohn's, previous bariatric surgery)
- Vegans and anyone who avoids oily fish, eggs and fortified foods
- People taking certain medications (some anticonvulsants, glucocorticoids, HIV therapies)
If two or more of these apply to you and you are considering dental implants, testing before surgery is a sensible step.
Understanding Your Vitamin D Blood Test Result
UK laboratories report 25(OH)D in nanomoles per litre (nmol/L). (US sources use ng/mL; multiply ng/mL by 2.5 to convert.) The reference bands most UK labs and NICE use are:
Vitamin D blood levels (nmol/L)
Serum 25(OH)D β UK reference bands
Deficient
Bone softening risk
Insufficient
Common in UK winter
Sufficient
NHS "adequate"
Optimal*
Often targeted pre-implant
*There is no universally agreed "optimal" level for implant surgery; many clinicians aim for at least 75 nmol/L. Levels above ~250 nmol/L can be harmful.
| 25(OH)D level | Classification | What it means |
|---|---|---|
| Below 25 nmol/L | Deficient | Risk of rickets/osteomalacia; treatment usually recommended |
| 25β50 nmol/L | Insufficient | May be inadequate for some people; supplementation advised |
| 50β75 nmol/L | Sufficient | Adequate for the general population |
| 75β125 nmol/L | Often described as optimal | Range many clinicians target before bone surgery |
| Above 250 nmol/L | Potentially toxic | Risk of hypercalcaemia β avoid |
Two points deserve emphasis. First, "sufficient" is defined around general bone health, not around the demands of surgical healing. Several implant and periodontal researchers argue for a higher target in patients about to undergo bone surgery β commonly at least 75 nmol/L β although there is no formal UK guideline stating this. Second, more is not better beyond a point: very high doses over long periods can push calcium levels dangerously high. Correction should be measured and, ideally, re-tested.
You can check your level with a simple venous blood sample. Our clinic offers a private vitamin D blood test with results from a UKAS-accredited laboratory, typically within a few working days. Results are sent to you as a laboratory report; we do not provide GP consultations, so any medical interpretation and prescribing should be discussed with your own GP.
Vitamin D and Gum Disease (Periodontitis)
Gum disease is the leading cause of tooth loss in adults, and it is also the single most important risk factor for peri-implantitis β the equivalent condition around dental implants. Understanding the vitamin D connection here therefore matters twice over.
The evidence
A substantial body of observational research links low vitamin D with more severe periodontitis. Large population datasets, including analyses of the US NHANES survey, show that people in the lowest vitamin D groups tend to have greater gum attachment loss and more bleeding on probing than those with higher levels. Systematic reviews and meta-analyses published in the last decade broadly support an association, although they also note that the quality of the trials varies and that association is not proof of cause.
Intervention studies are smaller but interesting. Some trials have found that patients who supplemented with vitamin D (often alongside calcium) during periodontal treatment showed better clinical attachment gain and reduced pocket depth than those who did not. Others have found no significant difference, so the picture is encouraging rather than conclusive.
Why it makes biological sense
Periodontitis is fundamentally an inflammatory response to bacterial plaque that spirals out of control and destroys bone. Vitamin D acts on both halves of that equation:
- Antimicrobial effect. Calcitriol switches on genes in gum cells and white blood cells that produce cathelicidin (LL-37) and Ξ²-defensins β natural antibiotics that kill periodontal pathogens such as Porphyromonas gingivalis.
- Anti-inflammatory effect. It reduces production of the pro-inflammatory cytokines (IL-1Ξ², IL-6, TNF-Ξ±) that drive bone-destroying osteoclast activity.
- Bone protection. By maintaining calcium supply and regulating bone turnover, it slows the loss of the alveolar bone that anchors your teeth.
For patients undergoing gum disease treatment at our practice, checking and correcting vitamin D is a low-cost, low-risk adjunct that may improve the response to hygiene therapy. It is not a replacement for professional cleaning and excellent home care β plaque control remains the foundation.
Vitamin D and Tooth Decay
The link between vitamin D and caries is older than most people realise. In the 1920s and 1930s, before fluoride toothpaste existed, several controlled studies in British and North American children found that supplementing with cod liver oil or exposing children to UV light reduced the number of new cavities. A 2013 systematic review revisiting these early trials estimated roughly a 50 % reduction in caries risk with vitamin D supplementation β a striking figure, though the studies were conducted in an era of widespread deficiency and would not meet modern trial standards.
Modern research continues to find associations. Children with low vitamin D in early life show higher rates of enamel defects (hypomineralisation) and early childhood caries. In adults the relationship is weaker but still present in several cohort studies.
The mechanism is straightforward:
- Enamel and dentine formation depend on adequate calcium and phosphate delivery during tooth development.
- Saliva quality β including its buffering capacity and antimicrobial peptide content β is influenced by vitamin D status.
- Immune surveillance in the mouth keeps cariogenic bacteria such as Streptococcus mutans in check.
For families, this is one more reason to follow the NHS advice on supplementing children from birth (drops for breastfed babies, 10 Β΅g daily for over-ones in autumn/winter). Our children's dentistry team is happy to discuss this alongside fluoride varnish and fissure sealants.
The Core Question: Does Vitamin D Affect Dental Implant Success?
This is the area generating the most research interest, and the one that matters most if you are planning treatment.
What happens when an implant is placed
A titanium implant does not simply sit in the jaw; it must osseointegrate β the bone must grow onto and into the microscopic surface of the implant until the two are structurally and functionally one. This process takes roughly three to six months and passes through predictable stages:
- Blood clot and inflammation (days 1β7). Platelets and immune cells arrive; growth factors are released.
- Woven bone formation (weeks 1β4). Osteoblasts lay down immature bone on the implant surface.
- Remodelling (weeks 4β12). Woven bone is replaced by dense lamellar bone; bone-to-implant contact increases.
- Maturation (months 3β6+). Bone continues to adapt to chewing loads.
Every one of those stages depends on osteoblast activity, calcium availability and controlled inflammation β the exact processes vitamin D regulates.
Osseointegration: bone bonding to the implant
Illustrative boneβimplant contact over the first 12 weeks
Schematic only, based on trends reported in animal and clinical studies. Individual healing varies widely.
What the research shows
Animal studies are consistent: vitamin D deficient animals show significantly lower bone-to-implant contact and poorer implant stability, and supplementation restores it. That establishes the biological plausibility.
Human evidence is growing. Key findings include:
- Early implant failure. Several retrospective studies of implant patients have found a higher rate of early failure (implants lost before loading, typically within the first few months) among patients with low serum vitamin D. One frequently cited series reported early failure rates roughly two to three times higher in deficient patients compared with those in the sufficient range. Other studies have found a trend in the same direction without reaching statistical significance, usually because early failure is a rare event and sample sizes are small.
- Implant stability. Studies using resonance frequency analysis (a way of measuring how firmly an implant is held) have reported higher stability values at the three-month point in patients with adequate vitamin D.
- Bone grafting outcomes. Where bone augmentation or sinus lifts are needed, adequate vitamin D appears to support better graft consolidation.
- Peri-implantitis. Low vitamin D has been associated with a higher prevalence of inflammation and bone loss around established implants, mirroring its relationship with periodontitis around natural teeth.
Systematic reviews published between 2019 and 2023 generally conclude that vitamin D deficiency is associated with increased early implant failure and that supplementation is a reasonable, low-risk measure, while calling for larger randomised trials to confirm causation. That is where the science honestly stands: strong plausibility, consistent direction of effect, and moderate-quality clinical evidence.
Putting it in perspective
Dental implants are already a highly successful treatment. Ten-year survival rates in healthy, non-smoking patients with good oral hygiene are typically quoted above 95 %. Vitamin D is not the difference between success and failure for most people. But it is one of a handful of modifiable risk factors β alongside smoking, uncontrolled diabetes, untreated gum disease and poor plaque control β that can be optimised before surgery at almost no cost. When the treatment itself is a significant investment, addressing those factors is simply good practice.
For a full picture of the procedure, healing and costs, see our dental implants guide and our dental implant cost breakdown.
Vitamin D, Bone Grafts and Sinus Lifts
Many patients who have been missing teeth for some time have lost bone volume and need a graft before or during implant placement. Grafted bone must be remodelled and replaced by the patient's own bone over several months, a process that is even more metabolically demanding than integrating an implant into native bone.
The research here is less extensive but points the same way: adequate vitamin D supports osteoblast recruitment and mineralisation of the graft, while deficiency has been associated with slower consolidation and greater graft resorption. If your treatment plan includes augmentation, correcting a low vitamin D level several weeks beforehand is particularly worthwhile.
When Risk Factors Stack Up: Smoking, Diabetes and Vitamin D
Risk factors for implant failure rarely act alone, and vitamin D deficiency tends to travel in company. Understanding how they interact helps explain why some patients heal effortlessly while others struggle.
Smoking
Smoking is the best-established modifiable risk factor for implant failure and peri-implantitis, roughly doubling the failure rate in most large series. Nicotine constricts the small blood vessels that deliver oxygen, nutrients and immune cells to the healing site, and the heat and chemicals in smoke directly impair osteoblast function. Smokers also tend to have lower vitamin D levels than non-smokers, partly through lifestyle and partly because smoking appears to alter vitamin D metabolism. A smoker with a low vitamin D level therefore faces a compounding effect on bone healing. Stopping smoking β even temporarily for the weeks around surgery β and correcting vitamin D together address two of the biggest controllable variables.
Diabetes
Poorly controlled type 2 diabetes impairs wound healing, reduces bone formation and increases susceptibility to infection. Well-controlled diabetes (HbA1c below around 53 mmol/mol, or 7 %) is generally not a barrier to implant treatment. There is a two-way relationship with vitamin D: deficiency is more common in people with diabetes, and low vitamin D is associated with poorer glycaemic control and greater insulin resistance. Some trials suggest that correcting deficiency modestly improves HbA1c. If you are diabetic and considering implants, testing both markers in the same blood draw is efficient and informative.
Osteoporosis and bisphosphonate medication
Osteoporosis itself does not rule out implants, but it does make bone quality assessment more important, and vitamin D adequacy is a cornerstone of osteoporosis management. If you take bisphosphonates (such as alendronic acid) or denosumab, tell your implant dentist β these drugs carry a small risk of medication-related osteonecrosis of the jaw after surgery, and your treatment plan will be adjusted accordingly. Vitamin D and calcium are routinely co-prescribed with these medications for good reason.
Gum disease history
Patients who have lost teeth to periodontitis are at higher risk of peri-implantitis around their replacement implants. The bacteria and the inflammatory tendency do not disappear when the teeth do. Because vitamin D influences both bacterial control and inflammation, maintaining adequate levels is part of a sensible long-term strategy for these patients, alongside meticulous maintenance with the hygienist.
The practical message: the more of these factors apply to you, the more valuable it becomes to optimise the ones you can control before surgery.
What Your Implant Dentist Assesses β and Where Vitamin D Fits
It helps to see where vitamin D sits in the overall assessment so it is neither ignored nor over-emphasised. A thorough implant consultation at our practice typically covers:
- Medical history and medications β diabetes, osteoporosis, immunosuppression, blood thinners, bisphosphonates, smoking and alcohol use.
- Clinical examination β gum health, plaque control, remaining teeth, bite and jaw joint function.
- 3D imaging (CBCT scan) β measures the height, width and density of bone available, and identifies nerves and sinuses that must be avoided.
- Periodontal charting β pocket depths and bleeding scores around remaining teeth; active gum disease is treated before implants are placed.
- Risk discussion β where modifiable risks are identified, we agree a plan to address them, which is where a vitamin D test may be recommended.
- Treatment planning β number and position of implants, whether grafting is needed, the type of final restoration, timeline and cost.
Vitamin D is not a routine test for every implant patient, and there is no UK guideline mandating it. We suggest it when risk factors are present, when bone grafting is planned, when a CBCT scan shows lower-density bone, or simply when a patient wants to leave as little to chance as possible. Because we can take the blood sample in the same building on the same day, the practical barrier is minimal.
Bone density on the CBCT scan
Dentists classify jaw bone from D1 (very dense, like oak) to D4 (soft, like polystyrene). Softer D3βD4 bone, common in the back of the upper jaw, achieves lower initial implant stability and relies more heavily on good bone formation during healing. In these sites the biological factors that support osteoblast activity β including vitamin D β arguably matter most. If your scan shows softer bone, that is a reasonable prompt to check your level.
Vitamin D and Healing After Extractions and Oral Surgery
Even outside implant dentistry, vitamin D influences how your mouth recovers. Extraction sockets heal through the same sequence of clot, woven bone and remodelling. Patients with adequate vitamin D tend to show better preservation of socket dimensions β relevant if you are planning an implant in that site later. Soft tissue healing also benefits from vitamin D's role in keratinocyte function and controlled inflammation.
If you are having a wisdom tooth removed or another extraction with a view to future implant placement, it is a good moment to check your level.
Vitamin D and Bruxism, TMJ and Bone Density
A few smaller associations are worth knowing about:
- Jaw bone density. Vitamin D deficiency contributes to systemic osteoporosis, which can reduce the density of the jaw and complicate implant planning in older patients.
- Bruxism (teeth grinding). Some studies have associated low vitamin D and low calcium intake with sleep bruxism, possibly through effects on muscle function and sleep quality. The evidence is preliminary, but if you grind your teeth it is one more reason to ensure adequate levels alongside a custom night guard.
- Burning mouth and oral discomfort. Nutritional deficiencies including vitamin D, B12 and iron are routinely checked in patients with unexplained oral burning.
How Much Vitamin D Do You Need?
UK official guidance
- All adults and children over 4: 10 Β΅g (400 IU) per day, with a supplement recommended from October to early March and year-round for at-risk groups.
- Babies under one: 8.5β10 Β΅g daily unless taking more than 500 ml of formula.
- Safe upper level: 100 Β΅g (4,000 IU) per day for adults. Do not exceed this without medical supervision.
Correcting a confirmed deficiency
If a blood test shows a level below 25 nmol/L, your GP may recommend a loading regimen β a higher dose over several weeks (for example, a total of around 300,000 IU spread over 6β10 weeks) followed by a maintenance dose. Levels in the 25β50 nmol/L range are usually managed with a standard daily maintenance dose of 20β50 Β΅g (800β2,000 IU) for a few months and a re-check.
Because dosing above the routine 10 Β΅g should be guided by a blood result and by your GP or pharmacist, we recommend testing first rather than guessing. High-dose self-medication over months without monitoring is unwise.
Food sources
Diet alone rarely provides enough in the UK, but it helps:
| Food | Approximate vitamin D per portion |
|---|---|
| Grilled salmon (100 g) | 10β15 Β΅g |
| Tinned sardines (100 g) | 5β8 Β΅g |
| Mackerel (100 g) | 8β13 Β΅g |
| Egg yolk (1 large) | 1β1.5 Β΅g |
| Fortified plant milk (250 ml) | 1.5β2 Β΅g |
| Fortified breakfast cereal (30 g) | 1β2.5 Β΅g |
| UV-exposed mushrooms (100 g) | 5β10 Β΅g |
Oily fish two or three times a week plus fortified foods can supply a meaningful share of your needs; a supplement covers the rest.
Timing and absorption
Vitamin D is fat-soluble, so taking it with a meal containing some fat improves absorption. Consistency matters more than time of day. Vitamin K2 and magnesium are often marketed alongside vitamin D; the evidence that they materially change dental outcomes is limited, but a balanced diet supplying both is sensible.
A Practical Pre-Implant Optimisation Plan
Here is how we suggest patients approach vitamin D when implant treatment is on the horizon. It is deliberately simple and fits around the standard planning timeline.
A simple pre-implant vitamin D plan
8β12 weeks before
Blood test for 25(OH)D (plus calcium, HbA1c if relevant)
6β10 weeks before
Correct any deficiency with supplements as advised; address smoking and gum health
2β4 weeks before
Optional re-test to confirm you are in range
Surgery day
Implant placed into optimised bone
3β6 months after
Osseointegration; maintenance dose continues
Always follow your implant dentist's individual advice β timings vary with the complexity of your case.
Step 1 β Test early (8β12 weeks before surgery)
Book a vitamin D blood test. If you have other risk factors, consider adding HbA1c (diabetes control), a full blood count and, for post-menopausal women or older men, discussing bone density with your GP. Your implant assessment appointment is the natural moment to plan this.
Step 2 β Correct if needed (6β10 weeks before)
If your level is below 50 nmol/L, discuss a correction regimen with your GP or pharmacist. Most people can raise their level meaningfully within six to eight weeks. Use this window to address the other modifiable risks too: stop smoking, complete any hygiene therapy needed to bring gum inflammation under control, and stabilise blood sugar if you are diabetic.
Step 3 β Confirm (2β4 weeks before)
An optional re-test confirms you are in the sufficient range. This is most valuable if your starting level was very low or if a bone graft is planned.
Step 4 β Surgery and healing
Continue a maintenance dose throughout the three-to-six-month integration period and beyond. Vitamin D is not a one-off pre-operative box to tick; ongoing adequacy supports the bone around your implant for life.
Step 5 β Maintenance for life
Keep up the autumn/winter supplement, attend regular implant maintenance appointments with the hygienist, and re-test every year or two if you were previously deficient.
Frequently Asked Questions
Can I still have a dental implant if my vitamin D is low?
In most cases yes, but your dentist may recommend correcting the deficiency first, particularly if a bone graft is planned or your level is very low. A delay of a few weeks to optimise healing conditions is usually a worthwhile trade-off.
Will taking vitamin D guarantee my implant succeeds?
No. Implant success depends on many factors: surgical technique, bone quality, gum health, smoking, diabetes, oral hygiene and the design of the final restoration. Adequate vitamin D removes one avoidable risk; it does not override the others.
How long does it take to raise vitamin D levels?
With an appropriate daily dose, most people see a meaningful rise within four to eight weeks. A GP-prescribed loading regimen for severe deficiency works faster. Levels then plateau, so ongoing maintenance is needed.
Should I take vitamin D2 or D3?
D3 (cholecalciferol) is generally preferred because it raises and sustains blood levels more effectively. Vegan D3 derived from lichen is widely available.
Can too much vitamin D harm my teeth or implants?
Excessive intake over long periods can cause hypercalcaemia, which is harmful to the kidneys and heart and does not benefit bone. Stay within the 100 Β΅g (4,000 IU) daily upper limit unless your GP advises otherwise, and do not take high doses without testing.
Does vitamin D help with receding gums?
Vitamin D supports gum tissue health and reduces inflammation, but it cannot regrow gum that has already receded. It may help slow further progression when combined with proper periodontal care. See our page on receding gums.
Is a vitamin D test included in an implant consultation?
Not automatically, but it is easily added. Our clinic runs private blood tests on site, so you can have the sample taken at the same visit as your dental assessment. You receive the laboratory report directly; we do not provide GP consultations, so any medical follow-up should be with your own GP.
Does sunlight through a window count?
No. Standard glass blocks the UVB wavelengths needed to make vitamin D. Office workers behind glass all day produce essentially none.
I take a multivitamin β is that enough?
Most multivitamins contain 5β10 Β΅g of vitamin D, which meets the basic UK recommendation but will not correct an existing deficiency. Check the label and test if in doubt.
Are there dental signs of vitamin D deficiency?
In adults there are no reliable oral signs; the condition is usually silent until bone problems appear. In children, severe deficiency can cause enamel defects and delayed tooth eruption. A blood test is the only dependable way to know your status.
Key Takeaways
- Vitamin D drives calcium absorption, bone remodelling and immune defence β all central to healthy gums, strong jaw bone and successful implant integration.
- Low vitamin D is extremely common in the UK, especially between October and March and among indoor workers.
- Evidence links deficiency with more severe gum disease, higher childhood decay rates and a higher risk of early dental implant failure.
- A simple blood test measures 25(OH)D. Many clinicians aim for at least 75 nmol/L before implant or bone graft surgery, although there is no universal target.
- Follow UK guidance: 10 Β΅g daily in autumn and winter for everyone, and correct confirmed deficiency under GP or pharmacist guidance before surgery.
- Vitamin D is one modifiable risk factor among several. Combine it with stopping smoking, controlling gum disease and excellent oral hygiene for the best long-term result.
How St Paul's Medical & Dental Can Help
Our City of London clinic beside St Paul's Cathedral combines an experienced implant team with an on-site private blood testing service. That means you can have your implant assessment and your vitamin D test on the same visit, receive your laboratory result within days, and plan any correction well before surgery.
- Book a dental implant consultation
- Book a private vitamin D blood test
- Gum disease treatment
- Dental hygienist appointments
Call 020 7183 3709 or book online. We are open Monday to Thursday 8amβ6pm and Friday 8amβ5pm, two minutes from St Paul's, Bank and Blackfriars stations.
References and Further Reading
- Scientific Advisory Committee on Nutrition (SACN). Vitamin D and Health. Public Health England, 2016.
- NHS. Vitamin D β Vitamins and minerals. nhs.uk (accessed September 2026).
- National Diet and Nutrition Survey (NDNS): results from years 9 to 11 (2016/17 to 2018/19). Public Health England, 2020.
- NICE Clinical Knowledge Summary. Vitamin D deficiency in adults. 2022.
- Hujoel PP. Vitamin D and dental caries in controlled clinical trials: systematic review and meta-analysis. Nutrition Reviews. 2013;71(2):88β97.
- Dietrich T, et al. Association between serum concentrations of 25-hydroxyvitamin D3 and periodontal disease in the US population. American Journal of Clinical Nutrition. 2004;80(1):108β113.
- Machado V, et al. Vitamin D and periodontitis: a systematic review and meta-analysis. Nutrients. 2020;12(8):2177.
- Mangano F, et al. Low serum vitamin D and early dental implant failure: is there a connection? A retrospective clinical study on 1740 implants placed in 885 patients. Journal of Dental Research, Dental Clinics, Dental Prospects. 2018;12(3):174β182.
- Werny JG, et al. Does vitamin D have an effect on osseointegration of dental implants? A systematic review. International Journal of Implant Dentistry. 2022;8:16.
- Guido Mangano F, et al. Is low serum vitamin D associated with early dental implant failure? A retrospective evaluation on 1625 implants placed in 822 patients. Mediators of Inflammation. 2016;2016:5319718.
- Javed F, et al. Efficacy of vitamin D3 supplementation on osseointegration of implants. Implant Dentistry. 2016;25(2):281β287.
- Bashutski JD, et al. The impact of vitamin D status on periodontal surgery outcomes. Journal of Dental Research. 2011;90(8):1007β1012.
Written by the clinical team at St Paul's Medical & Dental, 5 Ave Maria Lane, London EC4M 7AQ. This article reflects the published evidence as of September 2026 and will be reviewed annually.
Disclaimer
This article is for educational purposes only and is not a substitute for professional dental advice, diagnosis, or treatment. Individual conditions vary β please consult a qualified dental professional for personalised guidance. In a dental emergency, seek immediate professional care.
Next Review Due: September 2027








